Genetic Circuit & Parts

1. System Overview

We designed a dual-target "AND" gate detection system based on RNA aptamers for the simultaneous detection of NFL (neurofilament light chain) and GFAP (glial fibrillary acidic protein) in blood. The system consists of two plasmids:

When NFL and GFAP are both present, they bind to their respective aptamers on RNA1, causing RNA1 to dissociate from the T7 promoter on plasmid 2. T7 RNA polymerase then initiates sgRNA transcription. The sgRNA activates Cas13, which cleaves the reporter probe, ultimately producing a visible readout on a lateral flow test strip.

2. Mechanism

OFF State (No Target or Single Target Only)

Under arabinose induction, plasmid 1 transcribes RNA1 (containing the blocker sequence and dual aptamers). Through its T7 complementary sequence, RNA1 forms a stable duplex with the T7 promoter DNA on plasmid 2 (ΔG = -27.35 kcal/mol, RNAcofold modeling result), physically blocking T7 RNA polymerase binding. Transcription is terminated, and no sgRNA is produced. Cas13 remains inactive, and the reporter probe remains intact.

ON State (Both NFL and GFAP Present)

When both NFL and GFAP are present in the sample:

  1. NFL binds to the NFL aptamer on RNA1.
  2. GFAP binds to the GFAP aptamer on RNA1.
  3. Protein binding induces a conformational change in RNA1, causing it to dissociate from the T7 promoter DNA.
  4. T7 RNA polymerase binds to the promoter and transcribes sgRNA.
  5. sgRNA activates Cas13, which cleaves the reporter probe.

Test Strip Interpretation

State sgRNA Cas13 Activity Probe Status T Line C Line Interpretation
No target / Single target Absent Inactive Intact Visible Visible Negative (no dual target)
Dual target present Present Active Cleaved Invisible Visible Positive (NFL+GFAP both present)

Note: The C line serves as a control and should be visible in both positive and negative results; its absence indicates an invalid test strip.

3. Parts List

Part Type Source/Biobrick Function Reference
T7 promoterPromoterBBa_K5498011Drives transcription of RNA1 and sgRNAiGEM Registry
T7 terminatorTerminatorBBa_25ECYPMTTerminates transcriptioniGEM Registry
pBAD promoterPromoterBBa_25T3HMKAArabinose-inducible expression of RNA1iGEM Registry
NFL aptamer (MN734)AptamerTo be submittedSpecifically binds NFL protein, Kd = 8.1 nMMatsumoto et al., 2026
GFAP aptamerAptamerTo be submittedSpecifically binds GFAP protein, Kd = 0.62 μMCao et al., 2025
Blocker sequenceBlocking sequenceTo be submittedComplementary to T7 promoter, mediates RNA1 bindingTeam design
sgRNAReporter geneTo be submittedActivates Cas13, triggers signalTeam design
Cas13Effector proteinLiterature sourceCleaves RNA probe upon sgRNA activationLiterature

4. Plasmid Design

Plasmid Backbone Resistance Origin of Replication Insert Function
Plasmid 1 pUC19 Ampicillin pMB1 pBAD promoter - blocker - (G?S)? linker - GFAP aptamer - (G?S)? linker - NFL aptamer - T7 terminator Arabinose-inducible expression of RNA1
Plasmid 2 pUC57 Kanamycin pUC T7 promoter - sgRNA - T7 terminator Reporter plasmid, regulated by RNA1

Plasmid Maps

Plasmid 1 (pUC19):

Plasmid 1 map

Plasmid 2 (pUC57):

Plasmid 2 map

RNA1 Sequence: The full-length DNA sequence has been designed and is available in the Wiki appendix. It contains the T7 promoter complementary region, both aptamers, and linker sequences.

5. Workflow

  1. Sample Processing: Mix blood sample with detection reagents (containing Cas13, reporter probe, etc.), incubate at 37°C for 30 minutes.
  2. Apply to Test Strip: Apply the reaction mixture to the sample pad of a lateral flow test strip.
  3. Result Readout: Visually inspect after 5–15 minutes.
    • Positive (NFL+GFAP both present): C line visible, T line invisible.
    • Negative (no target or single target): Both C line and T line visible.
    • Invalid: C line invisible.

6. Key Supporting Data

Data Method Result Source
OFF-state binding energyRNAcofoldΔG = -27.35 kcal/molModeling
NFL aptamer affinityLiteratureKd = 8.1 nM (MN734)Matsumoto et al., 2026
GFAP aptamer affinityLiteratureKd = 0.62 μMCao et al., 2025
NFL aptamer–protein bindingHDOCK-288.23Modeling
GFAP aptamer–protein bindingHDOCK-301.91Modeling
"AND" gate validationODE modelingsgRNA produced only with both targetsModeling
Detection performanceODE modelingLOD = 0.57 pM, linear range 1–10 nMModeling

7. References

  1. Kim KY, Shin KY, Chang KA. GFAP as a Potential Biomarker for Alzheimer's Disease: A Systematic Review and Meta-Analysis. Cells. 2023;12(9):1309. (GFAP biomarker rationale)
  2. Wang X, Shi Z, Qiu Y, Sun D, Zhou H. Peripheral GFAP and NfL as early biomarkers for dementia: longitudinal insights from the UK Biobank. BMC Med. 2024;22(1):192. (NFL+GFAP combined detection rationale)
  3. Matsumoto M, Ikebukuro K, Tsukakoshi K. Competitive-SELEX discovery of DNA aptamers selective for neurofilament light chain in human plasma. Biochem Biophys Res Commun. 2026;796:153151. (NFL aptamer source)
  4. Buonocore J, et al. Plasma NfL and GFAP in the preclinical stages of neurodegenerative diseases: insights from the UK Biobank. J Neurol. 2025;272(12):755. (UK Biobank study)
  5. Cao Y, Lin Y, Chen L, et al. Screening of glial fibrillary acidic protein specific aptamer and application in the development of fluorescent biosensor based on isothermal amplification strategy. Chin J Anal Chem. 2025. doi:10.1016/j.cjac.2025.100184. (GFAP aptamer source)

8. Additional Notes